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Effect of Endurance Exercise and MitoQ Intake on Tau Hyperphosphorylation, Oxidative Stress, Antioxidant Modulating Factors, Mitochondrial Function, and Cognitive Function in Alcl3-Induced Alzheimer’s Disease Animal Model
Jin-Hue Jeong, Dong-Hun Choi, Jeong-Kook Lee, Joon-Yong Cho
Exerc Sci. 2021;30(3):396-406.   Published online 2021 August 31    DOI: https://doi.org/10.15857/ksep.2021.30.3.396

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Effect of Endurance Exercise and MitoQ Intake on Tau Hyperphosphorylation, Oxidative Stress, Antioxidant Modulating Factors, Mitochondrial Function, and Cognitive Function in Alcl3-Induced Alzheimer’s Disease Animal Model
Exercise Science. 2021;30(3):396-406   Crossref logo
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Neuroprotective Benefits of Exercise and MitoQ on Memory Function, Mitochondrial Dynamics, Oxidative Stress, and Neuroinflammation in D-Galactose-Induced Aging Rats
Brain Sciences. 2021;11(2):164   Crossref logo
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Inhibition of 2-Arachidonoylglycerol Metabolism Alleviates Neuropathology and Improves Cognitive Function in a Tau Mouse Model of Alzheimer’s Disease
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ALWPs Improve Cognitive Function and Regulate Aβ Plaque and Tau Hyperphosphorylation in a Mouse Model of Alzheimer’s Disease
Frontiers in Molecular Neuroscience. 2019;12:   Crossref logo
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Antia, a Natural Antioxidant Product, Attenuates Cognitive Dysfunction in Streptozotocin-Induced Mouse Model of Sporadic Alzheimer’s Disease by Targeting the Amyloidogenic, Inflammatory, Autophagy, and Oxidative Stress Pathways
Oxidative Medicine and Cellular Longevity. 2020;2020:1-14   Crossref logo
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Exercise-Induced Oxidative Stress and the Effects of Antioxidant Intake from a Physiological Viewpoint
Antioxidants. 2018;7(9):119   Crossref logo
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Mitochondria-specific antioxidant supplementation does not influence endurance exercise training-induced adaptations in circulating angiogenic cells, skeletal muscle oxidative capacity or maximal oxygen uptake
The Journal of Physiology. 2016;594(23):7005-7014   Crossref logo
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Deregulation of Mitochondrial Function: A Potential Common Theme for Cardiovascular Disease Development
Oxidative Stress in Aging. 2008;165-189   Crossref logo
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The Mechanism of Catalpol in Alzheimer’s Disease by Modulating Mitochondrial Function Through miR-124-Mediated STIM2
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Sodium butyrate protects against oxidative stress between obesity-prone and obesity-resistant rats induced by HFD through modulating Nrf2 pathway and mitochondrial function
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